Hypertension / diabetes complications – manageable!
High blood pressure (hypertension) is the most common and at the same time most underestimated sign of systemic heart and kidney disease, affecting around 1.5 billion people worldwide. In Germany, 20 to 30 million people are affected. Hypertension is caused by inflammation as a physical reaction to individual stress and/or environmental influences such as fine dust, chemicals and polluted food chains. If it persists, hypertension is a sign of a more serious disruption of the endothelium in the blood vessels and of fibrosis. According to current medical knowledge, both phenomena occur in parallel, including in the heart and kidneys. Causalities cannot be determined within this complex molecular system.
In the early stages of the disease, efficient medical treatments to prevent serious consequences such as heart attacks, dialysis and premature loss of years of life are neither possible nor have the diseases been able to be defined and detected early.Almost every diabetic also suffers from hypertension.
High blood pressure is the body's reaction to chronic inflammation and fibrosis. The immune system is activated "over the top". As the inner wall cells, the endothelium, become increasingly damaged, the inner wall of the arteries, for example, thickens, which in turn increases the pressure of the flow. At the same time, collagens increase outside the blood vessels. These have a stabilizing function and now themselves increase the pressure on the blood vessels from the outside.
The kidneys play a central role in regulating blood pressure; they filter the blood. If chronic inflammation and/or fibrosis are not successfully treated, endothelial damage spreads in the blood vessels. Fibrosis also spreads; normal tissue is replaced by scar tissue. The function of the kidneys continually decreases until this process can no longer be stopped. The current diagnostic and therapeutic dilemma.
The increased blood pressure damages the overtaxed heart, causing the heart muscles to thicken. The blood vessels in and around the heart narrow, which leads to a slowing of blood flow, a reduced supply (of oxygen, energy carriers, etc.) and a further increased load on the heart. Again, it is the damage to the endothelium and the fibrosis that continuously damage the organ. Without systemic and early analysis of this cellular protein-controlled system that controls high blood pressure and the cardiovascular-renal system, no efficient and successful treatment can be carried out.